ASBMB Today news story
Even when HIV is successfully suppressed, many people continue to experience neurological complications. Although antiretroviral therapy reduces HIV replication to undetectable levels, about 40% of people develop HIV-associated neurocognitive disorders. In a study from the Journal of Lipid Research, researchers at Temple University found that accumulation of Tat, an HIV protein involved in viral replication, disrupts organelle communication and lipid metabolism in neurons, offering a potential explanation for why these complications persist despite treatment.
Read the full news story here.
Source : ASBMB Today
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